ALVEOLAR BONE NECROSIS FOLLOWING HERPES ZOSTER INFECTION OF TRIGEMINAL NERVE: A CASE REPORT
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Abstract
Herpes zoster is a disease cause by varicella zoster virus (VZV). After the primary infection, which, causes chickenpox, the VZV remains latent in the sensory ganglia. The weakened immune systems, VZV can reactivate as herpes zoster (HZ), that made patient have Localized burning, prickling pain and painful vesicles on the skin and mucosa around the affected sensory nerve distribution. Oral manifestations appear when the maxillary or mandibular branches of trigeminal ganglia are involved. This report describes a case 65-year-old with underlying diabetes mellitus, hypertension and benign prostatic hyperplasia presented by exposure of right mandibular alveolar bone necrosis after Herpes zoster infection about 1 month. The Dental treatment plan was bone sequestrectomy. At follow-up revealed normal wound and bone healing. The pathogenesis of alveolar necrosis is still controversial. One hypothesis is that the alveolar artery may be compressed by the edema caused by the inflammation of the alveolar nerve. This process may result in ischemia of alveolar bone and cause bone necrosis.
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References
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